
Affiliated with a Castle Connolly Top Hospital
Who is Dr. Chu, Hematology / Oncology Specialist in Stanford, CA?
Dr. Gilbert Chu, MD is a Hematology / Oncology Specialist, who primarily practices in Stanford, CA with 1 additional practice location. He is board certified. Dr. Chu graduated from Harvard Medical School, Boston, Massachusetts and completed his residency at Stanford Univ Hosp, Medical Oncology Mass Gen Hosp, Internal Medicine. Dr. Chu is fluent in English, and is currently seeing new patients. Dr. Chu’s practice accepts Aetna, Cigna, UnitedHealthcare and other major insurance plans. To book an appointment or to confirm insurance options, please call Dr. Chu’s office at (650) 723-4000.
What are Areas of Expertise for Dr. Chu?
Dr. Gilbert Chu, MD is a highly-rated, board-certified Hematology / Oncology Specialist known for expertly diagnosing, treating, and managing a wide array of related conditions and procedures. Utilizing the latest medical advancements and evidence-based practices, Dr. Chu empowers patients to confidently navigate their health journey, specializing in Gastrointestinal Cancer, Unknown Primary Cancer, and comprehensive wellness support. Serving the Stanford, CA community, Dr. Chu is dedicated to enhancing lives through expert, patient-centered care.
Where did Dr. Chu go to medical school and complete their residency?
Fellowship: Stanford University School of Medicine,Stanford, Ca, United States
Residency: Stanford Univ Hosp, Medical Oncology Mass Gen Hosp, Internal Medicine | Massachusetts General Hospital
Medical School: Harvard Medical School, Boston, Massachusetts | Harvard Medical School, Boston, Massachusetts
Is Dr. Chu board certified as a Hematology / Oncology Specialist?
Yes, Dr. Gilbert Chu, MD is board certified by the American Board of Internal Medicine , American Board of Internal Medicine since 1983
What languages does Dr. Chu speak?
Dr. Chu and their clinical team can communicate with patients in the following languages:
English
What conditions does a Hematology / Oncology Specialist like Dr. Chu typically treat?
As a Hematology / Oncology Specialist, Dr. Chu diagnoses, treats, and manages a wide range of conditions. This condition information is derived from anonymized insurance claims and highlights the medical conditions most commonly treated by Dr. Chu. It provides insight into the doctor’s areas of experience and expertise based on real-world patient encounters from the past two years, updated quarterly. Learn more about our claims-based healthcare methodology.
Also known as:
- Colorectal Cancer
- Rectal cancer
- Colon cancer
- Bowel cancer
- Malignant neoplasm of rectum
ICD-10 Codes:
- C20: Malignant neoplasm of rectum
Also known as:
- Neuroendocrine Tumor
- Cancer
- Endocrine Disease
- Malignant NET
- Neuroendocrine Cancer
- NET Cancer
ICD-10 Codes:
- C7A8: Other malignant neuroendocrine tumors
What procedures does a Hematology / Oncology Specialist like Dr. Chu typically perform?
As a Hematology / Oncology Specialist, procedures performed by Dr. Gilbert Chu may include:
- 3D Conformal Radiotherapy
- Adaptive Radiation Therapy (ART)
- Adrenalectomy
- Blood Thinning Therapy / Anticoagulation
- Blood Transfusion Consultation
- Blood Transfusion Follow Up
- Bone Excision
- Bone Grafting
Please contact Dr. Chu's office directly to confirm care for your specific procedure, for more information, or to schedule a consultation.
Does Dr. Chu accept my insurance?
Dr. Chu accepts most major insurance plans. Important: Please call our office at (650) 723-4000 before your appointment to verify that your specific plan and network are accepted.
What insurance plans does Dr. Chu accept in Stanford, CA?
Dr. Chu in Stanford, CA accepts plans from many carriers. While this list is updated regularly, it is not a guarantee of coverage.
Top Insurances
Aetna
Blue Cross Blue Shield
Blue Cross Blue Shield of Illinois
Blue Cross Blue Shield of Michigan
Capital Blue Cross
Cigna
CMS
Harvard Pilgrim
Oscar
Oxford Health Plans
View All Insurances
Where is Dr. Chu's office located?
Recognitions
Publications
Cooperative Assembly of a Protein-D Filament for Nonhomologous End Joining
, 2013
Here Comes the Sun: Recognition of UV-Damaged D
, 2008
Cernunnos/XLF promotes the ligation of mismatched and noncohesive D ends
, 2007
Processing of D for nonhomologous end-joining is controlled by kise activity and XRCC4/Ligase IV
, 2007
Processing of D for nonhomologous end-joining by cell-free extract
, 2005
Toxicity from radiation therapy associated with abnormal transcriptiol responses to D damage
, 2004
Significance alysis of microarrays applied to the ionizing radiation response
, 2001
Electrophoretic mobility shift assays for protein-D complexes involved in D repair.
, 2012
An Information Theoretic
, 2011
Crystal structure of human XLF: A twist in nonhomologous D end-joining
, 2007
Electrophoretic mobility shift assays to study protein binding to damaged D.
, 2006
Assays for nonhomologous end joining in extracts
, 2006
Toxicity from radiation therapy associated with abnormal transcriptiol responses to D damage
, 2004
Portrait of transcriptiol responses to ultraviolet and ionizing radiation in human cells
, 2004
Class prediction by nearest shrunken centroids, with applications to D microarrays
, 2003
Interaction between UV-damaged D binding activity proteins and the c-Abl tyrosine kise
, 2002
Xeroderma pigmentosum complementation group E and UV-damaged D-binding protein
, 2002
Sypsis of D ends by D-dependent protein kise
, 2002
Diagnosis of multiple cancer types by shrunken centroids of gene expression
, 2002
Supervised learning from microarray data
, 2002
Activation of D-dependent protein kise by single-stranded D ends
, 2000
Structure of D-dependent protein kise: implications for its regulation by D
, 1999
Expression of the p48 xeroderma pigmentosum gene is p53-dependent
, 1999
The use of electrophoretic mobility shift assays to study D repair.
, 1999
Failure of hairpin-ended and nicked D to activate D-dependent protein kise
, 1998
p48 activates a UV-damaged-D binding factor
, 1998
D-dependent protein kise: D binding and activation in the absence of Ku
, 1998
Double strand break repair
, 1997
The end-joining reaction in V(D)J recombition.
, 1997
D-dependent protein kise is not required for accumulation of p53 or cell cycle arrest after D
, 1997
Ku86 defines the genetic defect and restores X-ray resistance and V
, 1996
Isolation of a cD encoding a UV
, 1996
Role of the Ku autoantigen in V(D)J recombition and double-strand break repair
, 1996
A NOVEL ROLE FOR D PHOTOLYASE
, 1994
RESTORATION OF X-RAY RESISTANCE AND V(D)J RECOMBITION IN MUTANT-CELLS BY KU CD
, 1994
XERODERMA-PIGMENTOSUM GROUP-E BINDING-FACTOR RECOGNIZES A BROAD-SPECTRUM OF D-DAMAGE
, 1994
INVOLVEMENT OF THE KU AUTOANTIGEN IN THE CELLULAR-RESPONSE TO D DOUBLE-STRAND BREAKS
, 1994
A D END-BINDING FACTOR INVOLVED IN DOUBLE-STRAND BREAK REPAIR AND V(D)J RECOMBITION
, 1994
CELLULAR-RESPONSES TO CISPLATIN - THE ROLES OF D-BINDING PROTEINS AND D-REPAIR
, 1994
MASSIVE CISPLATIN OVERDOSE BY ACCIDENTAL SUBSTITUTION FOR CARBOPLATIN - TOXICITY AND MAGEMENT
, 1993
PURIFICATION AND CHARACTERIZATION OF A HUMAN PROTEIN THAT BINDS TO DAMAGED D
, 1993
BAG MODEL FOR D MIGRATION DURING PULSED-FIELD ELECTROPHORESIS
, 1991
PULSED-FIELD ELECTROPHORESIS OF MEGABASE-SIZED D
, 1991
SEPARATION OF LARGE D BY A VARIABLE-ANGLE CONTOUR-CLAMPED HOMOGENEOUS ELECTRIC-FIELD APPARATUS
, 1991
CISPLATIN-RESISTANT CELLS EXPRESS INCREASED LEVELS OF A FACTOR THAT RECOGNIZES DAMAGED D
, 1990
XERODERMA PIGMENTOSUM GROUP-E CELLS LACK A NUCLEAR FACTOR THAT BINDS TO DAMAGED D
, 1988
Cooperative Assembly of a Protein-DNA Filament for Nonhomologous End Joining
, 2013
Local false discovery rate facilitates comparison of different microarray experiments
, 2009
Here Comes the Sun: Recognition of UV-Damaged DNA
, 2008
Cernunnos/XLF promotes the ligation of mismatched and noncohesive DNA ends
, 2007
Processing of DNA for nonhomologous end-joining is controlled by kinase activity and XRCC4
, 2007
Processing of DNA for nonhomologous end-joining by cell-free extract
, 2005
Toxicity from radiation therapy associated with abnormal transcriptional responses to DNA damage
, 2004
Significance analysis of microarrays applied to the ionizing radiation response
, 2001
Electrophoretic mobility shift assays for protein-DNA complexes involved in DNA repair.
, 2012
An Information Theoretic
, 2011
Crystal structure of human XLF: A twist in nonhomologous DNA end-joining
, 2007
Electrophoretic mobility shift assays to study protein binding to damaged DNA.
, 2006
Assays for nonhomologous end joining in extracts
, 2006
Toxicity from radiation therapy associated with abnormal transcriptional responses to DNA damage
, 2004
Portrait of transcriptional responses to ultraviolet and ionizing radiation in human cells
, 2004
Class prediction by nearest shrunken centroids, with applications to DNA microarrays
, 2003
Interaction between UV-damaged DNA binding activity proteins and the c-Abl tyrosine kinase
, 2002
Xeroderma pigmentosum complementation group E and UV-damaged DNA-binding protein
, 2002
Synapsis of DNA ends by DNA-dependent protein kinase
, 2002
Diagnosis of multiple cancer types by shrunken centroids of gene expression
, 2002
p53 binds and activates the xeroderma pigmentosum DDB2 gene in humans but not mice
, 2002
Supervised learning from microarray data
, 2002
Xeroderma pigmentosum p48 gene enhances global genomic repair and suppresses UV-induced mutagenesis
, 2000
Activation of DNA-dependent protein kinase by single-stranded DNA ends
, 2000
Structure of DNA-dependent protein kinase: implications for its regulation by DNA
, 1999
Expression of the p48 xeroderma pigmentosum gene is p53-dependent
, 1999
The use of electrophoretic mobility shift assays to study DNA repair.
, 1999
Failure of hairpin-ended and nicked DNA to activate DNA-dependent protein kinase
, 1998
p48 activates a UV-damaged-DNA binding factor
, 1998
DNA-dependent protein kinase: DNA binding and activation in the absence of Ku
, 1998
Double strand break repair
, 1997
The end-joining reaction in V(D)J recombination.
, 1997
DNA-dependent protein kinase is not required for accumulation of p53 or cell cycle arrest after DNA
, 1997
Xeroderma pigmentosum, Cockayne syndrome and trichothiodystrophy
, 1996
Ku86 defines the genetic defect and restores X-ray resistance and V
, 1996
Isolation of a cDNA encoding a UV
, 1996
Role of the Ku autoantigen in V(D)J recombination and double-strand break repair
, 1996
A NOVEL ROLE FOR DNA PHOTOLYASE
, 1994
RESTORATION OF X-RAY RESISTANCE AND V(D)J RECOMBINATION IN MUTANT-CELLS BY KU CDNA
, 1994
XERODERMA-PIGMENTOSUM GROUP-E BINDING-FACTOR RECOGNIZES A BROAD-SPECTRUM OF DNA-DAMAGE
, 1994
INVOLVEMENT OF THE KU AUTOANTIGEN IN THE CELLULAR-RESPONSE TO DNA DOUBLE-STRAND BREAKS
, 1994
A DNA END-BINDING FACTOR INVOLVED IN DOUBLE-STRAND BREAK REPAIR AND V(D)J RECOMBINATION
, 1994
CELLULAR-RESPONSES TO CISPLATIN - THE ROLES OF DNA-BINDING PROTEINS AND DNA-REPAIR
, 1994
MASSIVE CISPLATIN OVERDOSE BY ACCIDENTAL SUBSTITUTION FOR CARBOPLATIN - TOXICITY AND MANAGEMENT
, 1993
PURIFICATION AND CHARACTERIZATION OF A HUMAN PROTEIN THAT BINDS TO DAMAGED DNA
, 1993
BAG MODEL FOR DNA MIGRATION DURING PULSED-FIELD ELECTROPHORESIS
, 1991
PULSED-FIELD ELECTROPHORESIS OF MEGABASE-SIZED DNA
, 1991
SEPARATION OF LARGE DNA BY A VARIABLE-ANGLE CONTOUR-CLAMPED HOMOGENEOUS ELECTRIC-FIELD APPARATUS
, 1991
CISPLATIN-RESISTANT CELLS EXPRESS INCREASED LEVELS OF A FACTOR THAT RECOGNIZES DAMAGED DNA
, 1990
XERODERMA PIGMENTOSUM GROUP-E CELLS LACK A NUCLEAR FACTOR THAT BINDS TO DAMAGED DNA
, 1988